Alzheimer’s Disease
A progressive disease that destroys memory and other important mental functions.
Brain cell connections and the cells themselves degenerate and die, eventually destroying memory and other important mental functions.
Memory loss and confusion are the main symptoms.
No cure exists, but medications and management strategies may temporarily improve symptoms.
Cluster Number:
Wiki Number: W007
Diagnosis: Alzheimer’s Disease
US Patients:
World Patients: 30Mil
Sex Ratio: M; 3F
Age Onset: 65+to42% by 80+
Brain Area: hippocampus, amyloids & tau proteins, 19 genes
Symptoms: forgetting, poor short term memory, location confusion
Progression: poor thinking, repetitious conversations, abusive, anxious, paranoid, loses ability to live
Causes: 19 genes, head injuries, depression, hypertension, smoking
Medications: memantine, acetylcholinemesterase inhibitors
Therapies: Chess, book-reading, exercise;low-fat diet, caffeine, wine
Youtube Video: Alzheimer’s Disease-Early Signs
Amazon or Library Book:
Is It Alzheimer’s?
Amazon or Library Book:
36-Hour Day
Click the book to link or order from Amazon.
Click the book to link or order from Amazon.
Support Group: Alzheimer’s Association 800-272-3900
Contact your local Social Security office for possible Disability Benefits through their Disability Determination Services,
Section 12.02.
4 CURRENT ARTICLES
FROM PUBMED
The world-wide medical research
reports chosen for each diagnosis
Clicking each title opens the
PubMed article’s summary-abstract.
- Changes in iPSC-Astrocyte morphology reflect Alzheimer's disease patient clinical markersby Helen A Rowland on December 20, 2024
Human induced pluripotent stem cells (iPSCs) provide powerful cellular models of Alzheimer's disease (AD) and offer many advantages over non-human models, including the potential to reflect variation in individual-specific pathophysiology and clinical symptoms. Previous studies have demonstrated that iPSC-neurons from individuals with Alzheimer's disease (AD) reflect clinical markers, including β-amyloid (Aβ) levels and synaptic vulnerability. However, despite neuronal loss being a key hallmark...
- Biological brain age and resilience in cognitively unimpaired 70-year-old individualsby Anna Marseglia on December 20, 2024
INTRODUCTION: This study investigated the associations of brain age gap (BAG)-a biological marker of brain resilience-with life exposures, neuroimaging measures, biological processes, and cognitive function.
- Classifying Alzheimer's Disease Using a Finite Basis Physics Neural Networkby Logeshwari Dhavamani on December 20, 2024
The disease amyloid plaques, neurofibrillary tangles, synaptic dysfunction, and neuronal death gradually accumulate throughout Alzheimer's disease (AD), resulting in cognitive decline and functional disability. The challenges of dataset quality, interpretability, ethical integration, population variety, and picture standardization must be addressed using deep learning for the functional magnetic resonance imaging (MRI) classification of AD in order to guarantee a trustworthy and practical...
- Effect of bright-light therapy on depression and anxiety of a patient with Alzheimer's disease combined with sleep disorder: A case reportby Xi Mei on December 20, 2024
CONCLUSION: This case report suggested that bright light therapy can have a positive effect on sleep quality in elderly patients with AD and can be used as an effective and safe non-pharmacological treatment.