Disorder of Written Expression
Disorder of written expression is a type of learning disability in which a person’s writing ability falls substantially below normally expected range based on the individual’s age, educational background, and measured intelligence.
Cluster Number:
Wiki Number: W064
Diagnosis: Disorder of Written Expression
US Patients:
World Patients :
Sex Ratio:
Age Onset:
Brain Area:
Symptoms: writing disability where it falls substantially below normal expected range
Progression:
Causes: may also have dyslexia, dyscalculia (math difficulties) or behavior difficulties as learning difficulties
Medications:
Therapies: must support the child or adult positively to reduce fears of shame at previously-poor performances
Youtube Video: Disorder of Written Expression:
Living with Learning Disabilities
Amazon or Library Book: Handwriting Brain-Body Disconnect
Amazon or Library Book:
How to Write a Paragraph
Click the book to link or buy from Amazon.
Click the book to link or buy from Amazon.
4 CURRENT ARTICLES
FROM PUBMED
The world-wide medical research
reports chosen for each diagnosis
Clicking each title opens the
PubMed article’s summary-abstract.
- FOLR2+ macrophage depletion from intestinal metaplasia to early gastric cancer: single-cell sequencing insight into gastric cancer progressionby Yuxin He on December 20, 2024
CONCLUSIONS: Our work provides an understanding of the potential mechanisms underlying the malignant transformation of IM mediated by FOLR2^(+) macrophages.
- Depressed TFAM promotes acetaminophen-induced hepatotoxicity regulated by DDX3X-PGC1α-NRF2 signaling pathwayby Sisi Chen on December 20, 2024
CONCLUSIONS: This study demonstrates that depressed hepatic TFAM plays a key role in the pathogenesis of AILI, which is regulated by the DDX3X-PGC1α-NRF2 signaling pathway.
- The RRP9-JUN axis promotes breast cancer progression via the AKT signalling pathwayby Jinliang Huan on December 20, 2024
CONCLUSIONS: In conclusion, this study revealed the crucial role of RRP9 in BC progression and its probable novel mechanism, suggesting that RRP9 may be a promising candidate for the treatment of BC.
- SNAI1 promotes epithelial-mesenchymal transition and maintains cancer stem cell-like properties in thymic epithelial tumors through the PIK3R2/p-EphA2 Axisby Haoran E on December 20, 2024
CONCLUSION: The tumorigenic role of SNAI1 through the PIK3R2/p-EphA2 axis was preliminarily validated in TETs. A potential therapeutic strategy for TETs may involve the inhibition of SNAI1.